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MINI REVIEW
Year : 2017  |  Volume : 3  |  Issue : 3  |  Page : 96-100

Autophagy regulated by miRNAs in colorectal cancer progression and resistance


1 Department of Pathology, Stony Brook University, Stony Brook, NY, USA
2 Department of Pathology, Stony Brook University, Stony Brook, NY, USA; Department of Experimental Marine Biology, Key Laboratory of Experimental Marine Biology, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, Shandong, China
3 Shandong Academy of Pharmaceutical Sciences, Jinan, Shandong, China
4 Department of Pathology, Stony Brook University, Stony Brook, NY, USA; Shandong Academy of Pharmaceutical Sciences, Jinan, Shandong, China

Correspondence Address:
Jingfang Ju
Department of Pathology, Stony Brook University, Room 185, BST L9, Stony Brook, NY 11794, USA

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Source of Support: None, Conflict of Interest: None


DOI: 10.4103/ctm.ctm_64_16

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The catabolic process of autophagy is an essential cellular function that allows for the breakdown and recycling of cellular macromolecules. In recent years, the impact of epigenetic regulation of autophagy by noncoding miRNAs has been recognized in human cancer. In colorectal cancer, autophagy plays critical roles in cancer progression as well as resistance to chemotherapy, and recent evidence demonstrates that miRNAs are directly involved in mediating these functions. In this review, we focus on the recent advancements in the field of miRNA regulation of autophagy in colorectal cancer.


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